Iva Hafner Bratkovič
Department of Synthetic Biology and Immunology, National Institute of Chemistry, Ljubljana, SloveniaTalk: From Yeast Cells to Digital Twins: Charting Cellular Networks for Disease and Drug Discovery
Topic/subtopic: Molecular basis of disease and therapy or Cellular processes and signalling
Dr. Iva Hafner Bratkovič is a research professor at the National Institute of Chemistry, Slovenia. She received her PhD in 2008. During her postdoctoral work, she got engaged in the field of innate immunity, particularly in the mechanisms underlying neuroinflammation and NLRP3 inflammasome. She was a visiting scientist at the Technical University Munich and the Biomedical Research Institute of Murcia and a Fulbright research scholar at Boston Children’s Hospital/ Harvard Medical School.
Her group focuses on the mechanisms of NLRP3 inflammasome activation and its modulation, from basic molecular and cell biology to autoinflammatory disease models. She is also interested in the regulation of cell death, focusing on gasdermin D-driven pyroptosis. The major aim of her group is to reconstruct and rewire innate signaling and cell death pathways with synthetic biology tools for various applications.
Dr. Iva Hafner Bratkovič is a research professor at the National Institute of Chemistry, Slovenia. She received her PhD in 2008. During her postdoctoral work, she got engaged in the field of innate immunity, particularly in the mechanisms underlying neuroinflammation and NLRP3 inflammasome. She was a visiting scientist at the Technical University Munich and the Biomedical Research Institute of Murcia and a Fulbright research scholar at Boston Children’s Hospital/ Harvard Medical School.
Her group focuses on the mechanisms of NLRP3 inflammasome activation and its modulation, from basic molecular and cell biology to autoinflammatory disease models. She is also interested in the regulation of cell death, focusing on gasdermin D-driven pyroptosis. The major aim of her group is to reconstruct and rewire innate signaling and cell death pathways with synthetic biology tools for various applications.
